August 20, 2015—Professor Philip D. Rasico has been awarded the 25th International Ramon Llull Award (XXVè Premi Internacional Ramon Llull) in recognition of his research, teaching and promotion of the Catalan language. The award is made by the unanimous decision of a distinguished panel of scholars representing de Fundació Ramon Llull and the Congrés de Cultura Catalana. Professor Rasico is the first scholar from the United States to receive this recognition. The award will formally be made at a ceremony to be held in late 2015 in Andorra.
My favorite Polish photographer, , sent me his photo of the November 7 evening with Nobel laureate and , who is co-editor, with , of and translator of several volumes of his essays. And isn’t that room gorgeous? It’s the Americas Society in Manhattan. And don’t we all wish we lived in Manhattan? Except for the weather … and the traffic… and the noise…
J. F. Hocquette, S. Tesseraud, I. Cassar-Malek, Y. Chilliard, I. Ortigues-Marty. . (2007) Responses to nutrients in farm animals: implications for production and quality. 1.
Anna V. Tinker, Alex Boussioutas, David D.L. Bowtell. . (2006) The challenges of gene expression microarrays for the study of human cancer. 9, 333-339.
Paola Finocchietto, Fernando Barreyro, Silvia Holod, Jorge Peralta, María C. Franco, Carlos Méndez, Daniela P. Converso, Alvaro Estévez, Maria C. Carreras, Juan J. Poderoso, Alessandro Bartolomucci. . (2008) Control of Muscle Mitochondria by Insulin Entails Activation of Akt2-mtNOS Pathway: Implications for the Metabolic Syndrome. 3:3, e1749.
Is it a coincidence that the United States witnessed a dramatic rise in household debt in the years before the recession-that the total amount of debt for American households doubled between 2000 and 2007 to $14 trillion?
In this regard it is of interest that a common Gly482Ser polymorphism of the peroxisome-proliferator–activated receptor γ coactivator 1, a transcriptional regulator of genes responsible for mitochondrial biogenesis and fat oxidation, has been linked to an increased relative risk of type 2 diabetes in Danish populations as well as to altered lipid oxidation and insulin secretion in Pima Indians. In addition, two recent studies involving DNA-microarray analysis suggest that there is a coordinated reduction in the expression of genes encoding peroxisome-proliferator–activated receptor γ coactivator 1α, which are involved in oxidative phosphorylation, in the skeletal muscle of overweight patients with type 2 diabetes, obese Mexican-American patients with type 2 diabetes, and overweight nondiabetic subjects with a family history of diabetes.
To assess whether decreased mitochondrial activity may contribute to the increased intramyocellular lipid content, we also assessed the rates of muscle mitochondrial phosphorylation using 31P magnetic resonance spectroscopy. We found that the mitochondrial rates of ATP production were reduced by approximately 30 percent in the muscle of the insulin-resistant subjects, as compared with the insulin-sensitive control subjects. Consistent with this finding of altered mitochondrial function, we also found a reduced ratio of inorganic phosphate to phosphocreatine, which may reflect a lower ratio of type I fibers (mostly oxidative) to type II fibers (mostly glycolytic) in the insulin-resistant subjects. This finding is consistent with those of a biopsy study by Nyholm et al., who found an increased number of type IIb muscle fibers in overweight, insulin-resistant, first-degree relatives of patients with type 2 diabetes. Taken together, these data suggest that the insulin-resistant offspring of patients with type 2 diabetes have an inherited reduction in mitochondrial content in muscle, which in turn may be responsible for the reduced rates of mitochondrial oxidative phosphorylation.
Foreign debt is denominated in units of tradables and a liquidity constraint links credit-market access to the income generated in the nontradables sector and the relative price of nontradables.
Even if capital control/financial repression is otherwise inefficient, the government still has to rely on capital control/financial repression to raise revenue for public goods provision.
Fasting rates of glucose production were similar in the nine insulin-sensitive control subjects (2.3±0.1 mg per kilogram of body weight per minute) and the eight insulin-resistant subjects (2.0±0.3 mg per kilogram per minute, P=0.41) for whom results were available and were completely suppressed in both groups during the period of hyperinsulinemic–euglycemic clamping. In contrast, the rates of glucose infusion required to maintain euglycemia were approximately 60 percent lower in the insulin-resistant subjects than in the insulin-sensitive control subjects during clamping (3.3±0.3 mg per kilogram per minute vs. 7.7±0.5 mg per kilogram per minute, P). This reduction in peripheral glucose metabolism could be attributed mostly to a reduction of approximately 70 percent (P
We also show that trade linkages help explain cross-country correlations in exchange market pressure during crisis episodes, even after controlling for macroeconomic factors.